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hypertrophied

英 [haɪˈpɜːtrəʊfid]

美 [ˈhaɪpərˌtroʊfid]

网络  淋巴器官比正常时肥大; 肥大; 心肌肥大; 过度增大的

COCA.44733

英英释义

adj

  • (of an organ or body part) excessively enlarged as a result of increased size in the constituent cells
    1. hypertrophied myocardial fibers
    Synonym:enlarged

双语例句

  • Results: It was found that the myocardial chymase like activity increased markedly in hypertrophied rat ( 2,6,12 weeks);
    结果:术后2、6、12周心肌肥厚组大鼠心肌组织糜酶样活性较同期正常对照组显著增高(P<0.01);
  • Method: bFGF and TGF β 1 gene expression were detected by immunochemistry and in situ hybridization in 32 human hypertrophied heart ( right and left ventricle, each 16).
    方法:用免疫组织化学及原位杂交技术检测32例(左、右室心肌各16例)肥大心肌组织中bFGF和TGFβ1的表达。
  • Inhibition of Na+/ Ca+ exchange induces the suppression in systolic and diastolic function of myocardium, especially in hypertrophied heart.
    抑制心肌Na+/ca2+交换将导致心肌收缩和舒张功能降低,这种抑制作用在压力负荷型肥厚心肌表现更为显著。
  • Study on Ischemia-Reperfusion Injury in the Hypertrophied Myocardium
    肥厚心肌缺血再灌注损伤与心脏肾素
  • KB-R7943 completely blocked the activating effects induced by E-4031 in both of normal and hypertrophied cardiomyocytes.
    R7943可完全阻断正常组和肥厚组心肌细胞由E-4031诱导的激动作用。尼卡地平不能阻断E-4031的增加作用。
  • Kocher demonstrate the transplantation of stem cell can decrease apoptosis of hypertrophied myocytes in the peri-infarct region and improve cardiac function.
    Kocher等报道移植造血干细胞可改善心肌梗死周边区肥大心肌细胞的凋亡。因此提出干细胞不仅具有分化作用,也可以通过一些途径抑制损伤部位细胞凋亡的发生。
  • To examine changes of collagen network and Matrix Metaloproteinases ( MMPs) activity in hypertrophied rat myocardium.
    探讨压力负荷增高性心肌肥厚时心肌胶原网络及心肌细胞外基质金属蛋白酶(MMPs)活性的变化。
  • Effects of glucose metabolism mediated by PDH on apoptosis of hypertrophied cardiac myocytes during post-hypoxia reoxygenation
    PDH调控的糖代谢途径对缺氧后复氧的大鼠肥大心肌细胞凋亡的影响
  • These findings suggest that the protein synthetic activity was increased in the hypertrophied myocardial cells.
    本实验动物的心肌细胞,正处在工作负荷增加,蛋白质合成旺盛的心肌细胞肥大代偿期。
  • These results suggest that autophagy of cellular elements seems to play an important regulative role in hypertrophied process and dilated T-tubules actually reflect myocardial adaptive changes.
    这些结果说明,细胞成份的自体吞噬降解在心肌肥大过程中起着重要调节作用,扩张的T管也反映了心肌细胞外吐加快的适应性改变。